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Pathogenesis of Hepatitis B and Kidney Disease


2012-09-28 16:13

After diagnosed with Hepatitis B and Kidney Disease, patients often feel very worried, as they know little about this disease. How do they get HBV Kidney Disease? Read on to learn about the pathogenesis of this disorder.

What is Hepatitis B and Kidney Disease?

When HBV invade into the body, they will duplicate in the liver cells, leading to Hepatitis B. Besides, these viruses will combine with antibodies, producing immune complex. More and more immune complexes will circulate with the blood circulation and deposit in the renal tissue, causing pathological changes of glomerular basement membrane. With gradual damage of the renal tissues, kidney function declines gradually and symptoms will appear.

In mild cases, patients may present swelling on eyelids, lower back pain, fatigue, decreased urine output, etc. In severe cases, symptoms like high blood pressure, blood in the urine, swelling of limbs can be caused. Individuals with serious kidney damage will experience oliguria or anuria. In the end, Uremia can be caused.

Pathogenesis of Hepatitis B and Kidney Disease

The occurrence of Hepatitis B and Kidney Disease is confirmed to be associated with the immune injury caused by deposition of immune complex in the glomerular capillary loops. About 5 months after patient's affected by HBV, there may be HBcAb, HBeAb, and HBsAb in the blood serum. So these viruses are in free state for long time and they are very easy to be cracked.

Part of the polypeptide products are tested to be the antigenic determinant of HBsAb. These components will combine with antibodies, forming immune complex. With massive immune complex depositions in the glomerular capillary loops, complement can be activated, resulting in immune injury to the kidneys.




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